This has led to the suggestion that amylin can function as a neuropeptide in a similar manner to GLP-1, which has been reinforced by studies showing that the administration of compounds that inhibit the function of amylin leads to an increase in food intake in rodents 3
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Li Y, Perry T, Kindy MS, Harvey BK, Tweedie D, Holloway HW, Powers K, Shen H, Egan JM, Sambamurti K, Brossi A, Lahiri DK, Mattson MP, Hoffer BJ, Wang Y, Greig NH (2009 Jan 27) GLP-1 receptor stimulation preserves primary cortical and dopaminergic neurons in cellular and rodent models of stroke and Parkinsonism